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Medical Knowledge Base · Gynecology

Endometriosis

Classification, diagnosis, and treatment of a chronic inflammatory disease

Endometriosis is a chronic estrogen-dependent condition affecting approximately 10% of women of reproductive age. It manifests with a broad spectrum of symptoms — from chronic pelvic pain to infertility — and requires an individualized therapeutic strategy, medical or surgical, based on the extent of the disease and the patient's reproductive desire.

Prof. Violante Di Donato
Updated: April 2026
Sapienza University of Rome

Definition and Classification

Endometriosis is a chronic inflammatory condition defined by the presence of endometrial tissue outside the uterine cavity. The endometrium, which physiologically lines the interior of the uterus, is an estrogen-dependent tissue whose growth and activity are regulated by hormonal stimulation.

Endometriosis affects approximately 10% of women of reproductive age and tends to diminish after menopause. In many patients, it represents a significant cause of chronic abdominopelvic pain and may be associated with other comorbidities, including infertility.

Based on the location of ectopic endometrial tissue, genital and extragenital forms are distinguished.

Genital endometriosis

It may present in an internal form, termed adenomyosis, when endometrial tissue involves the myometrium, or it may affect other structures of the female reproductive system, including the ovaries, uterosacral ligaments, pouch of Douglas, vaginal fornices, portio, vagina, vulva, perineum, and round ligament.

Extragenital endometriosis

Located outside the reproductive tract, it may involve the bowel, diaphragm, and urinary tract, particularly the bladder and ureters.

Iatrogenic endometriosis

Consists of endometrial tissue located at surgical scars, following cesarean section or other gynecologic procedures.

Etiology

The etiology of endometriosis has not yet been definitively clarified; however, several pathogenetic hypotheses have been proposed.

Theory of retrograde menstruation

Proposes that endometrial cells from the uterine cavity may reach ectopic sites during menstrual flow, implanting on peritoneal and ovarian surfaces.

Coelomic metaplasia theory

Hypothesizes the development of endometrial or endometrial-like tissue outside the uterus, arising from other cell groups, under the influence of hormonal or immunologic factors.

Lymphatic-vascular transport theory

According to this hypothesis, endometrial cells may spread through the blood or lymphatic circulation, explaining rare distant locations such as diaphragmatic or pulmonary involvement.

Immune tolerance

Proposes that endometriosis may result from a reduced capacity for peritoneal clearance of endometrial cells from retrograde menstruation, related to alterations in normal immunologic mechanisms.

Symptoms

The clinical presentation of endometriosis is heterogeneous. In some cases the disease may be asymptomatic; more frequently it is associated with the following manifestations:

90%
Dysmenorrhea
(painful menstruation)
77%
Chronic pelvic
pain
76%
Dyspareunia
(pain during intercourse)
50%
Infertility

In extragenital forms, urinary symptoms may appear, such as pain during urination, urinary urgency, and renal failure in cases of urinary tract involvement, or bowel symptoms, such as diarrhea, tenesmus, rectal bleeding, and bowel obstruction in cases of intestinal involvement.

Symptom intensity does not necessarily correlate with disease extent: patients with severe endometriosis may present modest symptoms, while women with apparently mild forms may report highly debilitating complaints. Endometriosis is considered a disease with high social impact, due to its repercussions on quality of life, personal relationships, and productivity.

Diagnosis

The diagnosis of endometriosis is based on a thorough medical history, gynecologic examination, and imaging studies including ultrasound, MRI, CA125 plasma level measurement, and laparoscopy. The first-level diagnostic approach consists of a gynecologic examination combined with transvaginal ultrasound, which allows evaluation of reproductive tract involvement, such as the presence of ovarian cystic formations, and potential infiltration of adjacent structures.

If this examination is negative in the presence of strong clinical suspicion, MRI may be performed, useful for evaluating peritoneal implants, rectosigmoid endometriosis, the ileocecal region, and urinary tract lesions. The most accurate diagnostic method is laparoscopy, a minimally invasive surgical procedure that involves introducing instrumentation equipped with a camera into the abdominal cavity through small skin incisions, allowing both diagnostic confirmation and surgical treatment.

A potentially useful test is the measurement of CA125, a marker often elevated in patients with endometriosis. However, elevated values may also be found in other conditions, including malignancies and inflammatory conditions, which is why it is not used as a diagnostic test. It may have a role in follow-up, since serum levels may correlate with disease stage, treatment response, and identification of potential recurrences after medical or surgical treatment.

Staging classification

Endometriosis may be classified by severity into four stages:

I
Minimal
Few superficial implants, without significant adhesions.
II
Mild
Greater number of implants, deeper than stage I.
III
Moderate
Deep implants, ovarian endometriomas on one or both ovaries, and some thin adhesions.
IV
Severe
Numerous deep implants, large endometriomas on one or both ovaries, and dense adhesions.

Medical Therapy

Treatment choice depends on the severity of symptoms, the extent and location of the disease, the desire for pregnancy, and the patient's age. In cases of minimal or mild endometriosis, the primary indication is medical therapy, aimed at reducing estrogen production and, consequently, limiting both the growth and inflammatory activity of the endometriotic tissue.

1st Line
Progestins or combined estrogen-progestins
Result in a reduction of dysmenorrhea and chronic pain in 60–95% of cases. Approximately 25% of patients do not respond to treatment or experience side effects such as irregular bleeding, breast tenderness, nausea, and headache.
Evidence-Based Insights
Focus on the best medical therapy for endometriosis

Comparison of combined oral contraceptives, progestins, and intrauterine system — evidence from network meta-analyses and recent randomized trials

There is no definitive ranking of efficacy among different combined oral contraceptives (COCs) and different progestins for endometriosis, as available evidence shows overall similar efficacy among various formulations. However, some recent studies suggest relevant differences related to the type of estrogen used, tolerability, and therapeutic persistence.

Comparative evidence on efficacy

A network meta-analysis of 1,680 women showed that all COCs significantly reduce pelvic pain compared with placebo (mean reduction: 15.1 points on a 0–100 VAS scale), without substantial differences among formulations. Progestins also demonstrated a significant benefit. Results for the main options are:

OptionFormulationVAS Reduction (95% CI)
Levonorgestrel intrauterine system52 mg−17.7 points (−25.5 / −9.8)
Intramuscular progestinsMedroxyprogesterone acetate 150 mg every 3 months−13.2 points (−16.2 / −10.1)
Oral progestinsNorethindrone acetate, medroxyprogesterone acetate, dienogest, drospirenone−12.6 points (−15.3 / −9.8)

Ref.: [1]

Differences based on estrogen type in COCs

A 2025 comparative study showed that COCs containing natural estradiol (E2) or estetrol (E4) may be more effective than COCs with ethinylestradiol in treating chronic pelvic pain. Estetrol 15 mg/drospirenone 3 mg showed improvement similar to dienogest at 3 and 6 months, and superior to COCs with natural estradiol at 6 months. COCs with ethinylestradiol showed the most modest efficacy. A Cochrane review, however, concluded that direct comparative evidence between different COCs remains insufficient to establish a definitive hierarchy.

Ref.: [2, 3]

Comparisons between oral progestins

A 2025 randomized study of dienogest vs norethindrone acetate demonstrated that both effectively reduce pain (dysmenorrhea scores reaching 0.00 in both groups at 12 months). Norethindrone acetate 5 mg/day showed greater endometrioma reduction (p=0.037) and lower discontinuation rate (23.3% vs 47.5% at 6 months). An Italian study reported similar clinical satisfaction (71% vs 72%), but dienogest was better tolerated (80% vs 58%). In patients with ovarian endometriomas, dienogest showed greater symptom reduction. Compared with medroxyprogesterone acetate, dienogest demonstrated significantly superior pain reduction in some post-surgical series.

Ref.: [4, 5, 6, 7]

Dienogest — distinctive characteristics

Dienogest is among the most studied progestins in endometriosis. It increases PR-B receptor expression in endometriotic tissue, counteracting progesterone resistance. In several randomized studies it has shown efficacy comparable to GnRH agonists with a more favorable tolerability profile. A 2025 network meta-analysis identified it among the pharmacologic treatments with the most robust scientific support for endometriosis-related pain.

Ref.: [8, 9]

Levonorgestrel intrauterine system (LNG-IUS)

The LNG-IUS showed the greatest pain reduction in the network meta-analysis (−17.7 points). Long-term data document sustained efficacy up to 10 years, with 91.5% of patients maintaining treatment beyond 5 years (vs 21.9% with COCs or dienogest), fewer systemic side effects, and better therapeutic persistence. However, it is not the treatment of choice for ovarian endometriomas, as it does not consistently suppress ovulation.

Ref.: [1, 10]

Practical considerations

For COCs, continuous use is superior to cyclic use in pain control: residual dysmenorrhea 9.4% vs 20.9%, non-menstrual pelvic pain 9.4% vs 23.9%. Therefore, monophasic formulations are preferred. For progestins, the main differences concern tolerability, cost, route of administration, and persistence. Between 11% and 34% of patients do not respond to first-line hormonal therapies, due to possible progesterone resistance or intolerable side effects.

Ref.: [1, 8, 11]

Based on current evidence, COCs containing natural estradiol or estetrol represent an emerging prospect. Among progestins, dienogest and the levonorgestrel intrauterine system are the options with the most robust scientific support. Therapeutic choice must be individualized based on clinical presentation, disease phenotype, tolerability, and reproductive desire.

References 11 entries
1Endometriosis. As-Sanie S, Mackenzie SC, Morrison L, et al. JAMA. 2025;334(1):64-78. doi:10.1001/jama.2025.2975
2Comparative Study on the Effects of COC and Dienogest in Women With Endometriosis-associated Chronic Pelvic Pain. Caruso S, Cianci S, Caruso G, et al. Eur J Obstet Gynecol Reprod Biol. 2025;304:10-15. doi:10.1016/j.ejogrb.2024.11.015
3Oral Contraceptives for Pain Associated With Endometriosis. Brown J, Crawford TJ, Datta S, Prentice A. Cochrane Database Syst Rev. 2018;5:CD001019. doi:10.1002/14651858.CD001019.pub3
4Norethindrone Acetate Versus Dienogest for Pain Relief in Endometriosis — RCT. Gurbuz TB, Aslan K, Kasapoglu I, Muzii L, Uncu G. Eur J Obstet Gynecol Reprod Biol. 2025;310:113940. doi:10.1016/j.ejogrb.2025.113940
5Norethindrone Acetate or Dienogest for the Treatment of Symptomatic Endometriosis. Vercellini P, Bracco B, Mosconi P, et al. Fertil Steril. 2016;105(3):734-743. doi:10.1016/j.fertnstert.2015.11.016
6Dienogest or Norethindrone Acetate for the Treatment of Ovarian Endometriomas. Del Forno S, Mabrouk M, Arena A, et al. Eur J Obstet Gynecol Reprod Biol. 2019;238:120-124. doi:10.1016/j.ejogrb.2019.04.010
7Comparison of Dienogest With Medroxyprogesterone Acetate After Laparoscopic Surgery. Vahid-Dastjerdi M, Hosseini R, Rodi H, et al. Arch Gynecol Obstet. 2023;308(1):149-155. doi:10.1007/s00404-022-06898-2
8Endometriosis Is a Chronic Systemic Disease. Taylor HS, Kotlyar AM, Flores VA. Lancet. 2021;397(10276):839-852. doi:10.1016/S0140-6736(21)00389-5
9Pharmacologic Interventions for Endometriosis-Related Pain — Meta-Analysis. Kou L, Huang C, Xiao D, et al. Obstet Gynecol. 2025. doi:10.1097/AOG.0000000000005923
10Long-Term Efficacy and Safety of LNG-IUS as Maintenance Treatment for Endometriosis. Kim HY, Song SY, Jung SH, et al. Medicine. 2022;101(10):e29023. doi:10.1097/MD.0000000000029023
11Hormonal Drugs for the Treatment of Endometriosis. Capezzuoli T, Rossi M, La Torre F, Vannuccini S, Petraglia F. Curr Opin Pharmacol. 2022;67:102311. doi:10.1016/j.coph.2022.102311
2nd Line
GnRH agonists (leuprolide, triptorelin, goserelin, buserelin, nafarelin)
Induce pituitary desensitization with consequent suppression of ovarian estrogen production. In the initial phase they may cause a transient estrogen increase (flare-up) before suppression. Administration is injectable (IM, SC, parenteral, intravaginal) or via nasal sprays.
GnRH antagonists (elagolix, relugolix, linzagolix)
Competitively block pituitary GnRH receptors, with rapid reduction of gonadotropins and estrogens. Unlike agonists, they do not cause initial flare-up and act from the first dose. Available orally.
3rd Line
Aromatase inhibitors
Block peripheral estrogen synthesis. They are administered in combination with contraceptives or GnRH agonists. Main side effects include osteoporosis, vaginal dryness, nausea, and headache.
Symptomatic
NSAIDs
Useful for symptomatic pain control, but do not address disease mechanisms and do not modify its course.
Neuromodulators
Useful in selected patients with persistent chronic pain, particularly in the presence of a neuropathic component or central sensitization. They do not treat lesions or modify disease course but may contribute to refractory pain control.
Evidence-Based Insights
Focus on neuromodulators and supplements in endometriosis

Evidence on adjuvant therapies for chronic pelvic pain: from selective use of neuromodulators to the role of supplements

Neuromodulators

Neuromodulators are not a first-line therapy for endometriosis but may play a role in persistent pain phenotypes with a neuropathic or nociplastic component, or in chronic pelvic pain refractory to hormonal and/or surgical therapies. The biologic rationale is solid: a proportion of patients with endometriosis develop central sensitization that reduces response to lesion-directed therapies. In clinical practice, neuromodulators make most sense when pain appears "decoupled" from the cycle, with allodynia, hyperalgesia, overlapping pain comorbidities, or persistence after surgery.

Among the medications, amitriptyline, duloxetine, and venlafaxine show more favorable clinical signals, while the empiric use of gabapentin was strongly downgraded by the multicenter randomized GaPP2 trial (Lancet 2020), which showed no clinically relevant benefit in female chronic pelvic pain and recorded more adverse events compared with placebo. The SOGC 2024 guidelines recommend considering tricyclics for neuropathic pelvic pain and SNRIs in selected cases, but advise against gabapentin as a routine choice.

Overall evidence on neuromodulators in female chronic pelvic pain remains of low-to-moderate quality; the most favorable results come primarily from small-scale studies. Their use is appropriate in a multidisciplinary pain setting, not as standard endometriosis treatment.

Supplements

The ESHRE 2022 guidelines explicitly state that no specific nutritional intervention can be recommended to reduce pain or improve quality of life in women with endometriosis, due to insufficient and heterogeneous evidence. The main supplements studied are:

SupplementAvailable evidence
N-acetylcysteine (NAC)Most interesting clinical signal among supplements: some studies report pain reduction and endometrioma size decrease, but evidence quality remains low (small or observational studies).
Omega-3 / fish oilPossible modest benefit on pain in preliminary reviews, but the randomized SAGE trial (AJCN 2020) did not demonstrate a clear advantage over placebo.
Vitamin DSolid immuno-inflammatory rationale, contradictory clinical data; some trials report pain reduction, others results similar to placebo.
CurcuminStrong preclinical anti-inflammatory rationale; a triple-blind trial showed no clear benefit, while an add-on study to dienogest reported improvements in pain and quality of life.
Antioxidants (vit. C and E)2024 meta-analysis of RCTs: possible reduction of pain symptoms, but with strong heterogeneity and low-to-moderate certainty of evidence.

Neuromodulators: to be considered only in selected patients with suspected neuropathic component or persistent pain unexplained by anatomic burden; avoid routine empiric use of gabapentin. Supplements: none are recommended as standard of care; NAC, omega-3, and vitamin D remain adjuvant options to be discussed case by case, clarifying the limitations of the evidence. The strategy with the most robust evidence remains pain phenotype assessment, appropriate hormonal therapy, and a multidisciplinary approach.

Summary table
OptionPossible clinical roleEvidence qualityPractical message
Oral neuromodulators
(amitriptyline, duloxetine, venlafaxine)
Persistent pain with neuropathic/nociplastic component or refractory CPPLow–moderateIn multidisciplinary setting; gabapentin not routine
GabapentinDowngraded by GaPP2 trial (Lancet 2020); no relevant benefit in female CPPModerate (negative)Not recommended routinely; more AEs vs placebo
NACFavorable signal on pain and endometrioma sizeLowInteresting as add-on; not universally recommended
Vitamin DConflicting data; some positive studies, others similar to placeboLowCorrect documented deficiency; not as specific therapy
Omega-3 / fish oilPossible modest benefit on pain; SAGE trial does not confirmLowNot routinely recommended
Antioxidants (vit. C, E)2024 meta-analysis: possible pain reduction, strong heterogeneityLow–moderatePromising, but evidence still insufficient
References 13 entries
1ESHRE guideline: endometriosis. ESHRE Endometriosis Guideline Group. Hum Reprod Open. 2022;2022(2):hoac009. doi:10.1093/hropen/hoac009
2Guideline No. 445: Management of Chronic Pelvic Pain. Allaire C, Yong PJ, Bajzak K, et al. J Obstet Gynaecol Can. 2024;46(1):102283. doi:10.1016/j.jogc.2023.102283
3Chronic Pelvic Pain: ACOG Practice Bulletin No. 218. Obstet Gynecol. 2020;135(3):e98-e109. doi:10.1097/AOG.0000000000003716
4Gabapentin for chronic pelvic pain in women (GaPP2): multicentre, randomised, double-blind, placebo-controlled trial. Horne AW, Vincent K, Hewitt CA, et al. Lancet. 2020;396(10255):909-917. doi:10.1016/S0140-6736(20)31693-7
5The Effect of Neuromodulatory Drugs on Chronic Pelvic Pain in Women — Systematic Review. Andrade MA, Ferreira JAS, Nogueira AA, et al. Rev Bras Ginecol Obstet. 2022;44(9):891-898. doi:10.1055/s-0042-1756460
6The Role of Neuromodulation in Chronic Pelvic Pain. Patel CB, Kharin N, Lee D, et al. Pain Physician. 2022;25(4):E565-E580. PMID:35793177
7Efficacy of N-Acetylcysteine on Endometriosis-Related Pain, Endometrioma Size, CA125 and Fertility Outcomes. Anastasi E, Granato T, Falzarano R, et al. Int J Environ Res Public Health. 2023;20(6):5194. doi:10.3390/ijerph20065194
8Effects of Vitamin D Supplementation in Endometriosis — Systematic Review. Kalaitzopoulos DR, Samartzis N, Kolovos GN, et al. Reprod Sci. 2022. PMID:33508990
9Supplementation with vitamin D or omega-3 in adolescent girls with endometriosis (SAGE) — RCT. Nodler JL, DiVasta AD, Vitonis AF, et al. Am J Clin Nutr. 2020;112(1):229-236. doi:10.1093/ajcn/nqaa080
10Antioxidant supplementation on dysmenorrhea and endometriosis-associated pain — Meta-analysis of RCT. Baradwan S, Sendy F, Sendy S, et al. Obstet Gynecol Sci. 2024. PMID:38221738
11Vitamin C and E antioxidant supplementation in women with endometriosis — Meta-analysis. Bayu P, Meshram RJ, Fatimawati F, et al. 2024. PMID:38820340
12Dorsal root ganglion stimulation for chronic pelvic pain — retrospective review. Burns SL, Agarwal D, Hagedorn JM, et al. Neuromodulation. 2024. PMID:39239506
13Dorsal Root Ganglion Stimulation for Chronic Pelvic Pain Secondary to Endometriosis. da Silva Freitas T, et al. Neuromodulation. 2025. PMID:39729062

Surgical Therapy

Surgical therapy is reserved for cases of severe endometriosis, in patients unresponsive to pharmacologic therapy, in the presence of distorted pelvic anatomy, adhesions, bowel obstruction, or obstructive involvement of the urinary tract. The procedure is performed laparoscopically.

Treatment may initially be conservative, through cauterization of endometriotic foci, a procedure associated with an increase in fertility rates and a reduction in pain, although symptom recurrence is possible in some cases.

When the disease presents a severe picture, with persistent debilitating symptoms despite medical therapy and conservative surgery, definitive surgery may become necessary. This consists of removal of the uterus (hysterectomy), with preservation of the tubes and ovaries in women of reproductive age, or removal of the uterus, tubes, and ovaries (hysterectomy with salpingo-oophorectomy) in postmenopausal women.

Hysterectomy with salpingo-oophorectomy combined with complete excision of all endometriotic foci has a cure rate of 90%.

Endometriosis Pelvic Pain Laparoscopy Infertility Hormonal Therapy Definitive Surgery
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For information on gynecologic examination and the initial diagnostic pathway for endometriosis, visit the portal dedicated to clinical gynecology.
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VD
Prof. Violante Di Donato
Associate Professor of Obstetrics and Gynecology — Sapienza University of Rome
Gynecologic oncology surgeon, specialist in minimally invasive surgery and gynecologic oncology
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This article is for informational and educational purposes only. It does not constitute medical advice, diagnosis, or therapeutic recommendation. All clinical decisions must be based on individual assessment by a specialist physician. © 2026 Prof. Violante Di Donato.