violantedidonato.it Insights Uterine Fibroids
Medical Knowledge Base · Benign Uterine Pathology

Uterine Fibroids

Leiomyomas, myomas, and fibromatous uterus: diagnosis, minimally invasive surgical treatment, and fertility management

Uterine fibroids are the most frequent benign lesion of the uterus and affect a significant proportion of women of reproductive age. In most cases they remain asymptomatic; when they produce symptoms, effective and minimally invasive therapeutic options are now available, allowing uterine preservation.

Prof. Violante Di Donato
Updated: April 2026
Sapienza University of Rome
Uterine fibroids after multiple myomectomy — Prof. Violante Di Donato
Uterine fibroids after multiple myomectomy. Original surgical specimen — Prof. Violante Di Donato, Sapienza University of Rome.

Definition

Uterine fibroids represent the most frequent benign lesion of the uterus and develop predominantly during reproductive age. Also known as uterine leiomyomas or myomas, they are not generally associated with an increased risk of malignant uterine tumor, and only exceptionally are they linked to neoplastic disease.

Uterine fibroid size can vary considerably: from microscopic nodules invisible to the naked eye to large masses capable of distorting the uterus and significantly increasing its volume. They may be present as single formations or as multiple lesions. In the most advanced cases, the presence of numerous fibroids may cause uterine enlargement reaching high abdominal levels.

A significant proportion of women develop uterine fibroids during their lifetime. However, many patients remain asymptomatic and the diagnosis is frequently incidental during a gynecologic examination or routine pelvic ultrasound.

Symptoms and anatomical classification

Many women with uterine fibroids do not present obvious symptoms. When symptoms are present, their intensity depends mainly on the location, number, and size of the fibroids.

In symptomatic patients, the most frequent signs include:

Anatomical classification

From an anatomical standpoint, uterine fibroids are classified according to their site of development:

Evidence-Based Insights
Evidence-Based Insights Focus on FIGO Classification of Myomas

FIGO has established a numbering system from 0 to 8 to define the exact location of the myoma within the uterus. This distinction is the "gold standard" for the surgeon in choosing the operative instrumentation (e.g. hysteroscopic resectoscope vs laparoscopic or robotic approach).

FIGO classification of uterine leiomyomas — subclassification system 0–8

FIGO subclassification system for uterine leiomyomas (0–8). Red: submucosal (SM) · Green: intramural/subserosal · Yellow: hybrids

Grade Category Description
Submucosal Myomas (SM) — Grade 0–2
0 Submucosal Completely intracavitary — pedunculated
1 Submucosal <50% intramural (predominantly intracavitary)
2 Submucosal ≥50% intramural
Other Myomas (O) — Grade 3–8
3 Intramural Contacts the endometrium; entirely intramural (100%)
4 Intramural Purely intramural, without contact with endometrium or serosa
5 Subserosal Subserosal ≥50% intramural
6 Subserosal Subserosal <50% intramural
7 Subserosal Subserosal pedunculated (completely extracavitary)
8 Other locations Other locations: cervical, parasitic, broad ligament
Hybrid Myomas — e.g., 2-5
2-5 Hybrid Two numbers separated by hyphen: the first indicates the relationship with the mucosa, the second with the serosa. E.g., 2-5: ≥50% intramural on the endometrial side and ≥50% intramural on the serosal side.
Clinical implications for the choice of approach
  • Grades 0–2 (submucosal): hysteroscopic approach with resectoscope; grade 0 simpler, grade 2 requires greater experience
  • Grades 3–4 (intramural): laparoscopic, mini-laparotomic, or robotic myomectomy; grade 3 may require endometrial incision
  • Grades 5–7 (subserosal): laparoscopy, mini-laparotomy, or robotic; grade 7 pedunculated technically more favorable
  • Grade 8: approach to be evaluated case by case
  • Hybrids: dual approach (hysteroscopy + laparoscopy) often required in the same operative session

FIGO = Fédération Internationale de Gynécologie et d'Obstétrique. Int J Gynaecol Obstet. 2011;113(1):3-13.

Causes

The exact causes of uterine fibroids are not fully understood. However, several biological factors appear to contribute to the development of these lesions.

Fibroids are believed to derive from clonal proliferation of a single myometrial stem cell. Growth behavior may vary: some lesions grow slowly, others remain stable, and still others shrink spontaneously. During pregnancy some fibroids temporarily increase in size, but tend to decrease after delivery.

Risk factors

Complications

Although uterine fibroids are generally benign, they may cause clinically relevant symptoms. The most frequent complication is iron deficiency anemia due to heavy and prolonged menstrual bleeding, which may manifest with fatigue, weakness, and reduced functional capacity. Only rarely is the blood loss sufficient to require transfusion.

Malignant transformation of a benign fibroid into leiomyosarcoma is extremely rare. Studies report a prevalence of occult leiomyosarcoma in women operated for fibroids ranging from approximately 1 in 352 to 1 in 8,300 cases. Leiomyosarcomas generally represent distinct tumors that do not derive directly from fibroid degeneration.

Pregnancy and fibroids

In most cases, uterine fibroids do not prevent conception. However, certain locations, particularly submucosal fibroids, may interfere with embryonic implantation and increase the risk of infertility or miscarriage.

During pregnancy, the presence of fibroids may be associated with an increased risk of certain obstetric complications, including placental abruption, fetal growth restriction, and preterm birth. Assessment of these risks requires individualized specialist evaluation.

Evidence-Based Insights
Evidence-Based Insights Focus on Treatment Management
Initial diagnostic algorithm — Suspected uterine fibroids
Signs or symptoms suggestive of uterine fibroids
(heavy bleeding, compression symptoms, pelvic pain, reproductive problems)
Gynecologic history and physical examination including abdominal, speculum, and bimanual pelvic examination
Consider: urinalysis, TSH, complete blood count, vitamin D, pregnancy test
Is abnormal uterine bleeding the main symptom?
No
Yes
Proceed directly to ultrasound
Consider endometrial biopsy if risk factors for malignancy
Transabdominal and transvaginal ultrasound with Doppler
Uterine fibroids diagnosed — consider vitamin D supplementation if deficient
Symptomatic treatment
Heavy bleeding
Compression symptoms
NSAIDs, tranexamic acid, oral contraceptives, 52 mg levonorgestrel IUD
Gynecologic specialist evaluation
Lack of improvement or other indication → referral to gynecologist
Therapeutic algorithm — Symptomatic fibroids
Therapeutic options for symptomatic uterine fibroids
Patient seeking pregnancy
Patient not actively seeking pregnancy
Complete fertility evaluation
Other infertility factors → Reproductive endocrinologist
Severe symptoms without other infertility cause → Myomectomy or uterus-sparing treatment
Heavy menstrual bleeding
Volume or pain symptoms
Contraceptive steroids or tranexamic acid
Insufficient relief or symptom progression?
Oral GnRH antagonists or progesterone receptor modulators
Still insufficient?
Isolated fibroids (± adenomyosis)
Uterine artery embolization, radiofrequency ablation, ultrasound ablation
Insufficient relief → Hysterectomy with ovarian preservation
+ Diffuse adenomyosis / cervical dysplasia / uterus >24 weeks
Hysterectomy with ovarian preservation

Adapted from: Uterine Fibroids. N Engl J Med. 2024;391(18):1721-1733.

Treatment

Treatment of uterine fibroids must be personalized and depends on symptom severity, lesion size and location, patient age, and pregnancy desire.

Clinical observation

In cases where fibroids are small, asymptomatic, or do not cause clinically relevant consequences, periodic monitoring with clinical and ultrasound follow-up may be indicated. This approach is frequently adopted in patients approaching menopause.

Pharmacologic treatment

Some hormonal therapies may contribute to menstrual cycle regulation and bleeding reduction. GnRH agonists cause temporary hormonal suppression and may reduce fibroid size, generally as preoperative treatment. Oral GnRH antagonists may significantly reduce bleeding. Hormonal contraceptives and levonorgestrel intrauterine devices act primarily on symptom control.

Laparoscopic, mini-laparotomic, and hysteroscopic myomectomy

Myomectomy consists of selective surgical removal of fibroids with uterine preservation. It can be performed via laparoscopic, mini-laparotomic, or hysteroscopic approach depending on the location and size of the lesions. This procedure is indicated in women who wish to preserve fertility or retain the uterus. Laparoscopic and mini-laparotomic myomectomy are associated with reduced blood loss, shorter hospital stay, and faster recovery compared to traditional laparotomic surgery.

Laparoscopic Myomectomy Mini-laparotomy Hysteroscopy Fertility Preservation

Uterine artery embolization

Uterine artery embolization (UAE) is a non-surgical procedure performed in interventional radiology that reduces blood supply to fibroids, leading to volume reduction. It requires multidisciplinary evaluation with the gynecologist and interventional radiologist.

Hysterectomy

In the most complex cases or when conservative therapies are ineffective, removal of the uterus may be indicated. This is a definitive treatment that eliminates the risk of recurrence, but it is not appropriate for women who wish to have a future pregnancy.

Prevention

Current scientific knowledge does not allow identification of definitive preventive strategies for uterine fibroids. However, certain lifestyle habits may contribute to risk reduction: maintaining adequate body weight and a diet rich in fruits and vegetables appear to have a possible protective effect. Some studies suggest that the use of hormonal contraceptives may be associated with a lower incidence of uterine fibroids.

When to consult a physician
  • Persistent pelvic pain
  • Very heavy, prolonged, or painful menstruation
  • Bleeding between cycles
  • Difficulty emptying the bladder
  • Anemia symptoms (fatigue, paleness, weakness)
  • Significant vaginal bleeding or sudden-onset acute pelvic pain — urgent evaluation

Frequently asked questions

What is the difference between a fibroid, a myoma, and a fibromatous uterus?

Fibroid, myoma, and leiomyoma indicate the same benign formation of the uterus originating from the smooth muscle cells of the myometrium. The three terms are interchangeable in clinical practice. The term fibromatous uterus is used when multiple fibroids are present in the uterus simultaneously.

Ref.: [7, 16, 20]

Can a fibroid transform into a malignant tumor over time?

Malignant transformation of a benign fibroid into leiomyosarcoma is extremely rare. Studies report that the prevalence of occult leiomyosarcoma in women operated for suspicious fibroids ranges from approximately 1 in 352 to 1 in 8,300 cases. Leiomyosarcomas generally represent distinct tumors that do not derive directly from the degeneration of benign fibroids.

Ref.: [4, 5, 6]

In which cases is surgery strictly necessary and when is monitoring sufficient?

Surgery is indicated when:

  • Symptoms (heavy bleeding, pelvic pain, compression symptoms) significantly compromise quality of life
  • Anemia from chronic bleeding develops
  • Fibroids cause infertility or obstetric complications
  • The patient desires a definitive solution

Monitoring is appropriate when:

  • Fibroids are asymptomatic
  • The patient does not want intervention
  • The patient is in perimenopause (fibroids tend to regress after menopause)

Ref.: [7, 14, 16, 18]

Can fibroids cause infertility or complications during pregnancy?

The impact of fibroids on fertility depends primarily on their location. Submucosal fibroids are most frequently associated with infertility and miscarriage, as they may interfere with embryonic implantation and alter endometrial vascularization. Intramural fibroids may reduce fertility in some patients, while subserosal fibroids generally have little impact on reproductive capacity.

During pregnancy they may increase the risk of obstetric complications such as preterm birth, placental abruption, or fetal growth restriction.

Ref.: [8, 9, 10, 11]

What are the advantages of laparoscopic and mini-laparotomic myomectomy compared to traditional surgery?

Laparoscopic and mini-laparotomic myomectomy are associated with reduced intraoperative blood loss, shorter hospital stay, faster functional recovery, and less postoperative pain. Numerous studies and meta-analyses also demonstrate a reduction in complications compared to traditional laparotomic surgery.

Mini-laparotomy (4–7 cm incision) represents a valid alternative to laparoscopy, particularly for large fibroids or in cases of complex uterine anatomy, with comparable clinical outcomes and a more accessible learning curve.

However, situations remain in which traditional laparotomy may be preferable, for example in the presence of very voluminous fibroids, unfavorable location, or severe uterine anatomical distortion. The choice of approach must be personalized by the surgeon based on individual characteristics.

Ref.: [1, 12, 13, 18]

Are there effective pharmacologic therapies to shrink myomas without surgery?

Some medications may temporarily reduce fibroid volume and control symptoms. GnRH agonists may cause uterine volume reduction of up to 30–60% but are generally used for limited periods (3–6 months) as preoperative treatment, due to side effects from hypoestrogenism.

Oral GnRH antagonists (relugolix, elagolix) may significantly reduce bleeding with a more manageable side-effect profile. Hormonal contraceptives or levonorgestrel intrauterine devices act primarily on symptom control without reducing fibroid size.

Ref.: [7, 14, 15, 16]

What is the recurrence risk after surgical removal of a fibroid?

After myomectomy, the recurrence risk is not negligible. Studies indicate fibroid recurrence in 25% of cases within approximately 40 months and in 23–30% of cases within 5–7 years. The risk is greater in younger patients, in the presence of multiple fibroids, and in cases where the primary fibroid has not been completely removed.

For this reason, planning the timing of surgery — in relation to the patient's age and reproductive desire — is a central element of specialist consultation.

Ref.: [7, 17]

Is hysterectomy the only definitive solution for severe fibromatosis?

Hysterectomy represents the definitive treatment because it completely eliminates the uterus and therefore the risk of fibroid recurrence. However, it is not the only therapeutic option. In many patients, conservative strategies such as myomectomy, ablative techniques, and other procedures may be considered, depending on clinical characteristics and reproductive desires.

The decision must be shared between the patient and the surgeon, taking into account symptoms, age, fertility desire, and the anatomical characteristics of the fibroids.

Ref.: [7, 14, 18]

What happens to fibroids with the onset of menopause?

With menopause, there is a reduction in estrogen and progesterone production, hormones that stimulate fibroid growth. As a result, many fibroids tend to shrink spontaneously and symptoms — particularly heavy menstrual bleeding — resolve with cessation of menstruation.

If a fibroid continues to grow after menopause, urgent specialist evaluation is indicated, as this behavior is atypical and must rule out malignant pathology.

Ref.: [7, 16, 19]

Bibliographic references for FAQ 20 entries

Specialist Consultation Request

For evaluation of uterine fibroids, to discuss the indication for laparoscopic, mini-laparotomic, or hysteroscopic myomectomy, or for a surgical second opinion, you may request a consultation with Prof. Di Donato.

Book a consultation →
VD
Prof. Violante Di Donato
Associate Professor of Gynecology and Obstetrics — Sapienza University of Rome
Gynecologic oncology surgeon, expert in minimally invasive surgery and gynecologic oncology

This article is for informational and educational purposes only. It does not constitute medical advice, diagnosis, or therapeutic indication. Every clinical decision must be based on individual evaluation by a specialist physician. © 2026 Prof. Violante Di Donato.